Archives For Herpes

iStock_000006391407XSmallMany factors contribute to Cancer, but the key catalyst (even the underlying engine) is one of many Viruses, particularly CMV, but also VZV, Epstein Barr as well as other Viruses SV-40, BK, JCV and HPV. Each cancer in our body has one to many viruses associated with it. Some key ones are HPV – Human Papilloma Virus causes Cervical, Anal, Mouth, Throat, Lung, Vaginal and Penile Cancers. Epstein Barr causes Lymphoma, JC Polyoma Virus causes Colon Cancer. Epstein Barr, HPV & CMV have all been associated with Breast Cancer, thus one reason for it’s prevalence. Now down to kids Cancer, Medulloblastoma Neuroblastoma and it’s 99% terminal twin cancer Glioblastoma. For these cancers its mainly CMV (Human Cytomegalovirus), though other viruses like SV-40, JC and BK are also capable and have been found in these tumors.

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This is my latest research of my hypothesis that viruses cause cancer. This is in presentation form and I’ve also included the link to my key research backup for all that I can back-up from medical and scientific research. Appreciate your thoughts and considerations.

Gary Elsasser “Ayden’s Dad”

Viral Cancer Hypothesis Presentation

Research PDF

Viral Cancer Hypothesis Backup Research

https://www.dropbox.com/sh/6cgwg7cw1irxsui/2vBB71XDkL

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The human cytomegalovirus (HCMV) and glioma sym- posium was convened on April 17, 2011 in Washington, DC, and was attended by oncologists and virologists involved in studying the relationship between HCMV and gliomas. The purpose of the meeting was to reach a consensus on the role of HCMV in the pathology of gliomas and to clarify directions for future research. First, the group summarized data that describe how HCMV biology overlaps with the key pathways of cancer. Then, on the basis of published data and ongoing research, a consensus was reached that there is sufficient evidence to conclude that HCMV sequences and viral gene expression exist in most, if not all, malig- nant gliomas, that HCMV could modulate the malig- nant phenotype in glioblastomas by interacting with key signaling pathways; and that HCMV could serve as a novel target for a variety of therapeutic strategies. In summary, existing evidence supports an oncomodula- tory role for HCMV in malignant gliomas, but future studies need to focus on determining the role of HCMV as a glioma-initiating event.

HallmarksOfCancerList

https://www.dropbox.com/s/u0f80r60kc8qkh5/%28H%29%20Neuro%20Oncol-2012-Dziurzynski-246-55.pdf

“HCMV can also do every one of the things that generally considered the 10 hallmarks ofcancer,” says Kalejta, a member of the McArdle Laboratory for Cancer Research, Carbone Cancer Center, Stem Cell and Regenerative Medicine Center and Institute for Molecular Virology at UW-Madison.

http://www.med.wisc.edu/news-events/new-evidence-links-virus-to-brain-cancer/32922

iStock_000011406696Small“Typically, this pathway is only active during embryonic growth and development,” Melnick said, “but when CMV turns it back on, the resulting growth is a malignant tumor that supports production of more and more of the virus.”

http://dentistry.usc.edu/2011/11/14/common-virus-conclusively-identified-as-cancer-causing-oncovirus-ostrow-school-of-dentistry-researchers-pinpoint-cytomegalovirus’-central-role-in-salivary-gland-cancer/

Acute Cerebellar Ataxia – Often caused by Herpes Virus Infection in Cerebellum

Acute Cerebellar Ataxia can be caused by a Herpes Simplex Virus, often Varicella-Zoster Virus, the same that causes Chicken Pox & Shingles. The Virus can  infect the Cerebellum via catching the virus or via the vaccine per reports. When Acute Cerebellar Ataxia is caused by a virus it is specifically called Post Viral Cerebellar Ataxia. In addition, it has the exact same symptoms as Posterior Fossa Syndrome. This leads to the hypothesis that PFS & Post Viral Acute Cerebellar Ataxia may be one in the same.

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Ignition of a match, with smoke on dark backgroundCancers including Glioblastoma and Medulloblastoma over express the Cox-2 enzyme driving chronic inflammation and enabling the microenvironment for viruses like CMV and as well as cancer spread. Specific anti-inflammatories that target Cox-2 down regulation including Melatonin, Curcumin, Aspirin, Chokeberry/Aronia Berry and L-Lysine have been shown to help reduce cox-2 over expression.

https://www.dropbox.com/s/foz18g2jvs9i7gg/%28N%29%20HCMV-Cox2%20regulation.pdf

Brain cancers and head and neck cancers together account for more than 873,000 cases annually worldwide, with an increasing incidence each year.

A major problem with such research is that the role of many infectious agents may be underestimated due to the lack of or inconsistency in experimental data obtained globally.

Our analysis of the literature showed the presence of human cytomegalovirus (HCMV) in distinct types of brain tumour, namely glioblastoma multiforme (GBM) and medulloblastoma. In particular, there are reports of viral protein in up to 100% of GBM specimens.