“During the last years increasing evidence implies that human cytomegalovirus (CMV) can be attributed to human malignancies arising from numerous tissues. In this perspective, we will review and discuss the potential mechanisms through which CMV infection may contribute to brain tumors by affecting tumor cell initiation, progression and metastasis formation. Recent evidence also suggests that anti-CMV treatment results in impaired tumor growth of CMV positive xenografts in animal models and potentially increased survival in CMV positive glioblastoma patients. Based on these observations and the high tumor promoting capacity of this virus, the classical and novel antiviral therapies against CMV should be revisited as they may represent a great promise for halting tumor progression and lower cancer deaths.”
Archives For Brain Cancer
The following article explains how CMV or Cytomegalovirus recreates the Hallmarks of Cancer. The following are all signs of cancer: high prevalence of cell growth signaling, evading and disabling growth suppression, resisting cell death procedures, thus enabling cell immortality, building new blood vessels and enabling invasion and thus metastasis. Chronic inflammation also plays a role in cancer progression as well. Now see Hallmark by Hallmark how CMV can recreate all of these drivers that together enable cancer.
Starting off, Vitamin D is not a Vitamin, long time ago mis-namer. It’s actually a Pro-Hormone – a precursor building block to hormones and it’s unbelievably important. It acts as a molecular switch activating at least 200 target genes in our bodies, thereby regulating gene expression. And many of these are the ON switches for our immune system! I only wish I knew then, what I know now.
I’ve written often about the fact that 99% of Glioblastoma and 92% of Medulloblastoma show active CMV infections and show signs that CMV has hit and run certain tumor cells making genetic and epigenetic changes.
Now researchers are testing Neuroblastoma for CMV and the latest research shows 100% of the tumors showed signs of CMV or cyclomegalovirus. This continues to reinforce that brain cancers key consistent factor is the triggering of oncogenisis is by a viral infection that over expresses oncogenes and disables key anti-oncogenes thus driving these devastating cancers and leads towards potential opportunities for new and less devastating treatment options including using anti-virals like Valcyte.
This is my latest research of my hypothesis that viruses cause cancer. This is in presentation form and I’ve also included the link to my key research backup for all that I can back-up from medical and scientific research. Appreciate your thoughts and considerations.
Gary Elsasser “Ayden’s Dad”
Viral Cancer Hypothesis Presentation
Viral Cancer Hypothesis Backup Research
The human cytomegalovirus (HCMV) and glioma sym- posium was convened on April 17, 2011 in Washington, DC, and was attended by oncologists and virologists involved in studying the relationship between HCMV and gliomas. The purpose of the meeting was to reach a consensus on the role of HCMV in the pathology of gliomas and to clarify directions for future research. First, the group summarized data that describe how HCMV biology overlaps with the key pathways of cancer. Then, on the basis of published data and ongoing research, a consensus was reached that there is sufficient evidence to conclude that HCMV sequences and viral gene expression exist in most, if not all, malig- nant gliomas, that HCMV could modulate the malig- nant phenotype in glioblastomas by interacting with key signaling pathways; and that HCMV could serve as a novel target for a variety of therapeutic strategies. In summary, existing evidence supports an oncomodula- tory role for HCMV in malignant gliomas, but future studies need to focus on determining the role of HCMV as a glioma-initiating event.
“HCMV can also do every one of the things that generally considered the 10 hallmarks ofcancer,” says Kalejta, a member of the McArdle Laboratory for Cancer Research, Carbone Cancer Center, Stem Cell and Regenerative Medicine Center and Institute for Molecular Virology at UW-Madison.
Can the hospital recommend a dietary plan at the hospital for new cancer patients in addition to the current standard of care & clinical protocols since diet is not mentioned as part of most protocols? In this way, you are not deviating from standard protocols, just supporting healthy long term patient education on how to feed the body, not the cancer.Please read this and then consider the current standard care advice / plan of “have patients eat whatever they will eat on the hospital menu” in hospital after brain tumor resection surgery, radiation and beyond” versus the hospital designing, informing and offering a high good fat and low carbohydrate Ketogenic Diet” along with healthy dietary guidance for cancer patients under your care.